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Cellular ontogeny and MLL breakpoint site influence the capacity of MLL-edited CD34+ hematopoietic cells to initiate and recapitulate infant patients' features in pro-B-cell acute lymphoblastic leukemia (B-ALL). We provide key insights into the leukemogenic determinants of MLL-AF4+ infant B-ALL.

Original publication

DOI

10.1182/blood.2023020858

Type

Journal article

Journal

Blood

Publication Date

16/11/2023

Volume

142

Pages

1752 - 1756

Keywords

Infant, Humans, Gene Editing, Myeloid-Lymphoid Leukemia Protein, Precursor B-Cell Lymphoblastic Leukemia-Lymphoma, Hematopoietic Stem Cells, Oncogene Proteins, Fusion