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The data presented above suggest that one possible clinical use of TGF-beta would be to protect the bone marrow from the effects of myelosuppressive chemotherapeutic drugs by preventing entry or removing primitive stem cells from the cell cycle. It may also have the additional benefit of reducing the drug-induced neutrophil nadir by stimulating granulopoiesis. The availability of large quantities of recombinant TGF-beta will allow study of the pharmacokinetics with different routes of administration, dosage effects, and details of the pleiotropic effects on other cell systems. Experiments are in progress to determine whether TGF-beta will allow the delivery of higher amounts or more frequent doses of chemotherapeutic drugs and thus allow increased antitumor efficacy in tumor-bearing animals.

Original publication

DOI

10.1111/j.1749-6632.1991.tb17220.x

Type

Journal article

Journal

Ann N Y Acad Sci

Publication Date

1991

Volume

628

Pages

31 - 43

Keywords

Animals, Bone Marrow, Bone Marrow Cells, Cell Differentiation, Cell Division, Cytokines, Hematopoiesis, Hematopoietic Stem Cells, Humans, Transforming Growth Factor beta